statistiche ricercatore

Nome	#
The production of trophic factors of glial origin by guanosine involves mitogen-activated protein Kinase (MAPK) pathway and protects cultured neurons against NMDA excitoxicity.	101
Purine nucleosides protect injured neurons and stimulate neuronal regeneration by intracellular and membrane receptor-mediated mechanisms.	87
Intracellular accumulation of a mild-denatured monomer of the human PrP fragment 90-231, as possible mechanism of its neurotoxic effects.	80
Identification of a conserved N-capping box important for the structural autonomy of the prion alpha 3-helix: the disease associated D202N mutation destabilizes the helical conformation.	78
Characterization of the Proapoptotic Intracellular Mechanisms Induced by a Toxic Conformer of the Recombinant Human Prion Protein Fragment 90–231	76
Cultured Astrocyte Proliferation Induced by Extracellular Guanosine Involves Endogenous Adenosine and Is Raised by the Co-Presence of Microglia	75
Conformation dependent pro-apoptotic activity of the recombinant human prion protein fragment 90-231.	70
Recombinant human prion protein fragment 90-231, a useful model to study prion neurotoxicity.	70
ERK1/2 and p38 MAP kinases control prion protein fragment 90-231-induced astrocyte proliferation and microglia activation.	62
Differential role of EGF and BFGF in human GBM-TIC proliferation: relationship to EGFR-tyrosine kinase inhibitor sensibility.	62
Recombinant human prion protein fragment 90-231, a useful model to study prion neurotoxicity.	60
Totale	821

	Totale	Lug	Ago	Sett	Ott	Nov	Dic	Gen	Feb	Mar	Apr	Mag	Giu
2018/2019	37	0	0	0	0	0	0	0	0	0	0	0	37
2019/2020	153	47	11	0	9	6	15	20	12	3	19	11	0
2020/2021	96	12	0	11	1	6	17	1	0	14	12	16	6
2021/2022	44	1	0	0	15	0	0	1	3	2	1	6	15
2022/2023	205	12	29	13	28	23	40	9	13	34	1	1	2
2023/2024	58	10	2	5	1	1	22	16	0	0	1	0	0
Totale													821